We demonstrated that, in contrast to classical opioid receptors, ACKR3 won't bring about classical G protein signaling and isn't modulated because of the classical prescription or analgesic opioids, for example morphine, fentanyl, or buprenorphine, or by nonselective opioid antagonists like naloxone. Instead, we set up that LIH383, an ACKR3-selective https://billp788hzh4.wikistatement.com/user